Skip to main content
Chapters 38 total chapters

NEET UG · Biology

Evolution

Revise the concepts. Try a question. Understand your next step.

110 easy questions available · Try answers free. Sign in to save progress or take a quiz.

NEET UG · Biology

Continue studying Evolution

Understand the concept, practise questions and check your learning.

Understand

Calculate allele and genotype frequencies with explicit assumptions and explain how survival and reproductive contribution change a model population. Evolution: distinguish allele counts, equilibrium and selection

About 5 min · NEET UG

Read the guide

Practise

Try 110 easy chapter MCQs. Untimed; check answers and available explanations as you go.

Free answers · Sign in to save progress

Practise chapter MCQs

Check your learning

Prepare a timed chapter quiz. Choose the available question count and pace before starting.

Sign-in required · Explanations after submission

Prepare a chapter quiz

This is chapter practice, not a full-length NEET mock paper.

Choose your practice

All Biology chapters →

These are playable question counts after quality and duplicate checks.

How practice and quizzes work

The practice player uses single-correct-answer MCQs. Chapter quizzes are not full-length NEET mock papers. Check the practice guide and official syllabus sources.

Revise before you practise

Study resources 48

Open full resource hub →

Evolution: distinguish allele counts, equilibrium and selection

Calculate allele and genotype frequencies with explicit assumptions and explain how survival and reproductive contribution change a model population.

What you will understand

For NEET UG Biology students: count alleles separately from genotypes, use Hardy-Weinberg as a stated model and connect heritable variation to survival and reproductive contribution.

Jump to chapter practice →

Quick answer: a population frequency is not an individual adaptation

Population genetics tracks allele and genotype frequencies. A standard diploid individual has its own genotype; it does not change into the population’s new frequency. Hardy-Weinberg gives a comparison model under random mating and no mutation, migration, selection or drift in the ideal large-population setting. Departures need investigation rather than an automatic claim that one particular force caused them.

Combine the two mixed cells into one heterozygote class

A square of side five hundred pixels is split in each direction into 350 and 150 pixel intervals, representing gamete allele probabilities p=0.7 and q=0.3. Areas are AA 0.49, Aa 0.21, aA 0.21 and aa 0.09. The two mixed cells combine into heterozygote frequency 2pq=0.42; they are not two different unphased genotype classes. This is an ideal random-union model, not observed population data. Hardy-Weinberg equilibrium assumptions and simple autosomal diploidy are stated in the lesson. The picture does not infer which allele is dominant.
The common square splits each gamete axis into p = 0.7 and q = 0.3. Areas are 0.49 AA, 0.21 Aa, 0.21 aA and 0.09 aa. The two mixed cells represent the same unphased heterozygous genotype and add to 0.42. This is an ideal random-union account, not a plot of observed population data or a claim about which allele is dominant.

Open full-size diagram ↗

Take a square root only when the model permits it

In a stated Hardy-Weinberg model with complete recessive expression, 9% of individuals have phenotype aa. Here q2=0.09q^2=0.09, so q = 0.3 and p = 0.7. Heterozygote frequency is 2pq = 0.42, not 0.09 and not 0.3. The inference depends on the model and on the phenotype correctly identifying aa; a phenotype proportion alone is not always an allele-frequency estimate.

Allele counting does not require Hardy-Weinberg proportions

For a sample of diploid autosomal genotypes, count two A copies from AA and one from Aa, then divide by twice the number of individuals. This estimates the sample allele frequency even if genotype proportions do not match equilibrium expectations. The frequency equation and the equilibrium prediction answer different questions: one counts observations, while the other predicts under assumptions.

Separate an observed survival step from the next generation

An original clonal population begins with one hundred individuals of each of two heritable variants R and S. In the specified environment sixty R and twenty S survive, changing the R fraction from one half to three quarters. If every survivor makes two clonal descendants, the next group contains 120 R and 40 S and retains the three-quarter fraction. The model excludes new mutation and migration during these steps and specifies equal output after survival. It does not show individuals acquiring a needed mutation, claim that survival alone guarantees reproductive success, or represent measured field data.
The original clonal model begins with 100 of each heritable variant. Sixty R and twenty S survive; R now makes up three-quarters. If every survivor produces two clonal descendants, the next group contains 120 R and 40 S. No mutation or migration is added in this model. It illustrates a mechanism, not a field experiment or proof that survival alone always determines reproductive success.

Open full-size diagram ↗

Count alleles before comparing with an equilibrium model

An original sample contains 40 AA, 40 Aa and 20 aa individuals.

p=2(40)+402(100)=0.60,p=\frac{2(40)+40}{2(100)}=0.60,
and q = 0.40. If Hardy-Weinberg expectations are then calculated, AA, Aa and aa frequencies would be 0.36, 0.48 and 0.16. The observed 0.40, 0.40 and 0.20 differ from those expectations. That comparison alone does not identify selection, sampling effects or another specific cause.

Selection acts on existing heritable differences in context

A variant can contribute more descendants in one environment without being universally superior. Mutation supplies new genetic variants; selection can change how existing heritable differences contribute to later generations. Genetic drift can also change frequencies through chance, especially in small populations. A frequency shift is therefore not evidence that organisms produced a needed mutation because they tried to adapt.

Keep a fraction and a survival rate separate

In the illustrated model, R survival is 60/100 = 60%, while S survival is 20%. Among the eighty survivors, however, R frequency is 60/80 = 75%. The 60% survival rate and 75% survivor composition have different denominators. Equal clonal output after survival preserves the 75% composition in the next group; it does not create another increase merely because reproduction occurred.

Use the equilibrium equations with the right scope

For two alleles, p+q=1p+q=1 is the frequency total. Under the stated equilibrium model, p2+2pq+q2=1p^2+2pq+q^2=1 describes genotype proportions. The first identity does not itself establish equilibrium. Dominance affects phenotype mapping, while the genotype-frequency calculation depends on the mating and population assumptions.

Common mistakes: confuse copies, individuals and denominators

Ask whether the numerator counts allele copies, genotypes, survivors or descendants. State the total used for each fraction. A recessive phenotype can support a square-root inference only in the appropriate model. Do not label every change natural selection without evidence about heritable differences and differential contribution.

Try it: recover the heterozygote frequency

A stated Hardy-Weinberg two-allele model has aa frequency 0.16. Find q, p and the expected Aa frequency.

Reveal the answer

q is the square root of 0.16, or 0.4; p is 0.6. The expected Aa frequency is 2 × 0.6 × 0.4 = 0.48. The 0.16 is a genotype frequency, while 0.4 is an allele frequency. These values follow the supplied equilibrium assumptions.

Try it: infer a mechanism cautiously

A small isolated population changes allele frequency between generations, but no survival or reproductive advantage has been demonstrated. Does the frequency change alone prove natural selection?

Reveal the answer

No. Chance sampling and drift, among other possibilities depending on the conditions, can change frequencies. Evidence about differential reproductive contribution associated with heritable traits is needed to support a selection explanation. A change and its cause are different claims.

Come back tomorrow: compare two kinds of counting

Count A copies in a genotype sample without assuming equilibrium, then calculate equilibrium expectations as a separate step. Revisit crosses in Inheritance and Variation, or connect target-specific effects to Biotechnology Applications.

The practice action opens the broad evolution chapter pool, not a Hardy-Weinberg-only set.

Sources and review

Technical review: Codex (automated), 24 October 2026. Core mechanisms were compared with the linked primary textbooks and university or government references. All diagrams, problem inputs and explanations are original. Numerical balances and diagram directions are checked separately. Independent human subject review is not claimed. These focused guides support the existing chapter catalogue; they do not claim complete current exam or syllabus coverage.

Choose your next chapter task

For a numerical error, name the numerator and denominator. For a mechanism error, separate observed frequency change from evidence about why it occurred.

Open chapter practice and quiz choices →

Sources and reading
Type at least 2 characters or jump directly:
🔓

Save your learning progress

Sign in with a free account to save your practice answers and quiz results, then return to them when you revise.